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Methamphetamine is indicated as an integral part of a total treatment program for Attention Deficit Hyperactivity Disorder ADHD which typically includes other remedial measures psychological, educational, social for a stabilizing effect in children over 6 years of age with a behavioral syndrome characterized by the following group of developmentally inappropriate symptoms: moderate to severe distractibility, short attention span, hyperactivityemotional labilityand impulsivity. The diagnosis of this syndrome should not be made with finality when these symptoms are only of comparatively recent origin. Non-localizing soft neurological s, learning disabilityand abnormal EEG may or may not be present, and a diagnosis of central Meth drug interactions system dysfunction may or may not be warranted.

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Aims: Methamphetamine is an amphetamine derivative that is abused increasingly world-wide at an alarming rate over the last decade. Pre-clinical and human studies have shown that methamphetamine is neurotoxic to brain dopamine and serotonin. Other lines of study indicate that stress enhances the vulnerability to drug abuse. The purpose of this review is to shed light on the biochemical similarities between methamphetamine and stress in an effort to highlight the possibility that prior exposure to stress may interact with methamphetamine to exacerbate neurotoxicity.

Methods: A review of the Meth drug interactions on methamphetamine and stress was conducted that focused on the common neurotoxic and biochemical consequences of methamphetamine administration and stress exposure. : Experimental findings of a large of studies suggest that there are parallels between stress and methamphetamine with regard to their ability to increase glutamate release, Meth drug interactions a metabolic compromise and cause oxidative damage. Please enable it to take advantage of the complete set of features!

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Methamphetamine

Abstract Aims: Methamphetamine is an amphetamine derivative that is abused increasingly world-wide at an alarming rate over the last decade. Similar articles Chronic stress augments the long-term and acute effects of methamphetamine.

Matuszewich L, Yamamoto BK. Matuszewich L, et al. PMID: Methamphetamine-induced alterations in monoamine transport: implications for neurotoxicity, neuroprotection and treatment. Volz TJ, et al. PMID: Review.

Structural and metabolic brain changes in the striatum associated with methamphetamine abuse. Chang L, et al.

Methamphetamine-induced neural and cognitive changes in rodents. Marshall JF, et al. Altered energy production, lowered antioxidant potential, and inflammatory processes mediate CNS damage associated with abuse of the psychostimulants MDMA and methamphetamine.

Downey LA, et al.

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Eur J Pharmacol. Epub Jan See all similar articles.

Almalki AH, et al. Healthcare Basel.

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Role of nitric oxide in psychostimulant-induced neurotoxicity. Bashkatova V, Philippu A. Bashkatova V, et al. AIMS Neurosci. Brown CN, et al.

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Front Psychiatry. The interactions of dopamine and oxidative damage in the striatum of patients with neurodegenerative diseases. Li H, et al. J Neurochem.

Methamphetamine interactions

Epub Nov 4. Serial exposure to ethanol drinking and methamphetamine enhances glutamate excitotoxicity.

Blaker AL, et al. Epub Oct See all "Cited by" articles. Publication types Research Support, N. Research Support, U. Gov't, Non-P.

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